ISSN: 2578-4676
Authors: Pati P*
The tradition of imbibing alcoholic beverages can be traced back to ancient civilizations. Alcohol abuse is a major contributor to mortality, societal issues, and a strain on the healthcare system in the United States. Alcohol abuse impacts more than 29 million individuals and results in over 140,000 deaths each year in the United States. Long-term alcohol consumption is a well-established factor that increases the risk of tissue damage. Both acute and chronic alcohol intake can adversely affect renal function, particularly in the presence of hepatic illness. Scientists have observed that alcohol can cause alterations in the structure and function of the kidneys, leading to a decreased ability to maintain proper fluid and electrolyte balance in the body. Chronic drinkers may suffer from decreased amounts of vital electrolytes in their blood and significant disturbances in the body’s acid-base equilibrium. In addition, drinking can disrupt the hormonal processes that regulate kidney function. Persistent alcohol consumption worsens liver disease, causing additional injury to the kidneys by compromising their ability to balance salt and fluid levels, which might potentially result in sudden kidney failure. The correlation between chronic alcohol intakeD and kidney damage is fascinating yet contentious, as the molecular pathways behind alcohol-induced kidney impairment remain little comprehended. Existing epidemiological research have yielded ambiguous results, and there is a scarcity of experimental evidence that directly establishes a causal relationship between alcohol use and kidney injury. This review investigates the potential correlation between chronic alcoholism and the risk of renal injury.
Keywords: Alcohol Abuse; Alcohol Intake; Kidney Injury; Oxidative Stress
