ISSN: 2574-7770
Authors: Mina TK
In a previous editorial we’ve demonstrated that Increasing evidence points to the cytotoxicity of islet amyloid polypeptide aggregates showing them not just an insignificant phenomenon derived from the disease progression but as agents which directly induce processes that impair the functionality and the viability of beta-cells eventually leading to the loss of beta-cell mass in type 2 diabetes (T2D) patients.
Keywords: Amyloid Deposits; Cancer; Type 2 Diabetes Mellitus
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