Ahmad completed a Ph.D. in Molecular Genetics from University of Bundelkhand, Jhansi, India. He completed his first postdoctoral training at the Department of Microbiology and Immunology at the Indiana University School of Medicine in Indianapolis. where his research focused on understanding how interaction of HIV-1 (AIDS virus) envelope glycoproteins with host cell receptors lead to membrane fusion and viral entry. He completed his second postdoctoral training from Texas Tech University, El Paso Texas where his research focused on the mechanisms of genetic resistance to HIV-1 infection. Later he joined University of Texas at El Paso Texas as Scientist and lecture. Where he thought cellular Immunology to Master student and subsequently, carried his research on T cell-mediated immune control of persistent HIV infection, for the purpose of vaccine design. He joined SOUTHERN RESEARCH, as scientist , a well-known Pharma's Company of United States. Here the overall goal of my project is to identify new therapies that target influenza virus replication. A primary concern with the current drugs (amantadines and neuraminidase inhibitors) used to treat influenza is the development of resistance mutations that negate therapeutic benefit. We hypothesize that compounds that specifically target the polymerase complex might reduce the frequency of escape mutations, or promote escape mutants that are unfit for replication. Additionally, here he is working independently on Zika project. Zika virus (ZIKV) poses a serious public health threat due to the possibility of rapid global spread of the virus and its link to congenital birth defects such as microcephaly (MCPH), which is a serious neurodegenerative disease with lifelong consequences. Apoptotic death of ZIKV-infected fetal neural stem/progenitor cells (NSCs) seems to be a key factor that leads to the loss of neurons from cerebral cortex and the consequent fetal neurodegeneration seen in MCPH. Currently, the underlying molecular mechanism by which ZIKV induces apoptosis of NSCs is poorly understood. They have obtained very interesting evidence indicating that ZIKV causes suppression of the host cell AKT and mTOR proteins, which are involved in negative regulation of apoptosis. In addition to that, He is the member of Board of Director Integrated Biotechnological Research Institute; and is a member of several editorials boards and reviewer for scientific journals. He has also delivered papers to several international conferences.
Other Editorial Board Members
Eugene A Permyakov
Institute for Biological Instrumentation of the Russian Academy of Sciences
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